Introduction One of the most characteristic language disorders, specific and striking what is called dysphasia. Some authors prefer ‘or Disfasoideo dysphasic syndrome. Long is their history and names that have received over the years, depending on the age and nationality of the authors. Thus in 1888 there was talk of idiopathic Alalia Audimudez to refer to the Dysphasia. Names In the following century denominations abounded Congenital Aphasia, Aphasia Child Idioglosia, Simple Delay, Noise and Deafness Verbal Agnosia to describe the same phenomenon. In English literature refers to “Specific Developmental Expressive Dysphasia Tipe (Developmental Dysphasia Type Specific Expressive) while American authors use the terms of Aphasia Infantile or Congenital. Not only the names abound, but the definitions of the table, among which we highlight the following over the years: 1. Lack of speech in a child with normal hearing, average intelligence and no known brain injury (Nadoleczny, 1926).
2. Disturbance of internal language (Descoeudres, 1936). 3. Specific dysfunction in the development of the expression and / or reception of speech and language in the absence of other disabilities that may be considered as possible causes, such as hearing impairment, deficits in peripheral structures of speech, mental retardation, personality disorder, injury brain or psychotic disorders (Benton, 1964). 4.
Idiopathic disorder, not organic in the acquisition of speech by alterations in the structures is in charge of the collection, integration and conceptualization of language. The real Dysphasia must not show any motor impairment, affective, sensory, sensory or mental (1979). 5. Failure of normal language development can not be explained in terms of mental or physical, hearing impairment, of emotional disturbance or environmental deprivation (Bishop, 1992). 6. It is a perturbed form of language organization that builds on the foundation at odds with the common language. The dysphasic children living in a linguistic framework of its own and that once established, is difficult to undo. Therefore the dysphasic child is not only a poor speaker, but a child whose personality has been structured in any particular manner, according to a troubled relationship. As we have seen there are multiple definitions for a single box, although most agree that the disorder must be pure in the sense of not having any other associated pathology that can be interpreted as the cause of the problem, ie, the specific care the table. Moreover, all posit that the language would be the only factor altered. Concluding the above idea, considering the Dysphasia as a specific language impairment, would be assuming in the first place that has no apparent cause and secondly what would be affected would be only the language. However, following the first could hypothesize that point is not that there is no causality but the etiology is not passive case-finding by conventional methods of assessment such as electroencephalography, Audio-impedance analysis, CT, etc, and its cause could be found, for example, functional brain changes and dynamics that affect higher nervous activity as Azcoaga posit. Being even more inquisitive, we could argue that every phenomenon is a causality that causes it, regardless whether or not the means of passive case-finding.